找到约 9 条 “Recombinant Human AR (E.Coli)” 相关结果 (用时 0.107 秒)

目录号 产品名称 中文名称 靶点
M58346 Recombinant Human PPARG (E.Coli) 重组人PPARG (E.Coli) Target Proteins
PPARG蛋白是一种核受体,它是脂肪细胞分化的关键调节因子,能够促进脂肪细胞的生成和脂肪积累。同时,它还参与调节脂肪酸的β-氧化和葡萄糖代谢,对维持能量平衡和胰岛素抵抗等生理过程具有重要影响。PPARG蛋白还能够抑制NF-κB介导的促炎反应,从而发挥抗炎作用。PPARG蛋白还参与调节肿瘤细胞的增殖、凋亡和侵袭等恶性行为。
M58721 Recombinant Human AR (E.Coli) 重组人AR (E.Coli) Target Proteins
Steroid hormone receptors are ligand-activated transcription factors that regulate eukaryotic gene expression and affect cellular proliferation and differentiation in target tissues
M58812 Recombinant Human PPARA (E.Coli) 重组人PPARA (E.Coli) Target Proteins
Ligand-activated transcription factor. Key regulator of lipid metabolism.
M58813 Recombinant Human PPARD (E.Coli) 重组人PPARD (E.Coli) Target Proteins
Ligand-activated transcription factor key mediator of energy metabolism in adipose tissues 
M58828 Recombinant Human SARS2 3CLpro (E.Coli) 重组人SARS2 3CLpro (E.Coli) Target Proteins
主要蛋白酶(Mpro),也称为3-胰凝乳蛋白酶样蛋白酶(3CLpro),是β冠状病毒中一类高度保守的半胱氨酸水解酶
M58834 Recombinant Human SMARCA2 (E.Coli) 重组人SMARCA2 (E.Coli) Target Proteins
Involved in transcriptional activation and repression of select genes by chromatin remodeling (alteration of DNA-nucleosome topology).
M58875 Recombinant Human ARID1B (E.coli) 重组人ARID1B (E.coli) Target Proteins
ARID1B is a DNA-binding subunit of the Brahma-associated factor chromatin remodelling complexes, which play a key role in the regulation of gene activity. The function of remodelling complexes can be regulated by their subunit composition, and there is some evidence that ARID1B is a component of the neuron-specific chromatin remodelling complex. This complex is involved in the regulation of stem/progenitor cells exiting the cell cycle and differentiating into postmitotic neurons.
M58876 Recombinant Human ARNT (E.coli) 重组人ARNT (E.coli) Target Proteins
Required for activity of the AHR.
M58895 Recombinant Human SARM1 (E.coli) 重组人SARM1 (E.coli) Target Proteins
NAD+ hydrolase, which plays a key role in axonal degeneration following injury by regulating NAD+ metabolism








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